Blar i forfatter "Rismo, Renathe"
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Association of High-Density Lipoprotein Cholesterol With GFR Decline in a General Nondiabetic Population
Melsom, Toralf; Norvik, Jon Viljar; Enoksen, Inger Therese; Stefansson, Vidar Tor Nyborg; Rismo, Renathe; Jenssen, Trond; Solbu, Marit Dahl; Eriksen, Bjørn Odvar (Journal article; Tidsskriftartikkel; Peer reviewed, 2021-05-18)<i>Introduction</i> - Although lower high-density lipoprotein cholesterol (HDL-C) levels are considered a risk factor for cardiovascular disease (CVD), experimental evidence suggest that aging, inflammation, and oxidative stress may remodel HDL-C, leading to dysfunctional HDL-C. Population studies on HDL-C and loss of the glomerular filtration rate (GFR) reported inconsistent results, but they used ... -
Ernæringstilstanden hos pasienter innlagt i en indremedisinsk avdeling. Identifisering og oppfølging av pasienter med ernæringsforstyrrelser
Togstad, Lene; Rismo, Renathe (Master thesis; Mastergradsoppgave, 2004) -
Loss of interleukin 33 expression in colonic crypts- a potential marker for disease remission in ulcerative colitis
Gundersen, Mona Dixon; Goll, Rasmus; Hol, Johanna; Olsen, Trine; Rismo, Renathe; Sørbye, Sveinung Wergeland; Sundnes, Olav; Haraldsen, Guttorm; Florholmen, Jon (Journal article; Tidsskriftartikkel; Peer reviewed, 2016-10-17)Interleukin 33 (IL-33) is a cytokine preferentially elevated in acute ulcerative colitis (UC), inferring a role in its pathogenesis. The role of IL-33 in intestinal inflammation is incompletely understood, with both pro-inflammatory and regulatory properties described. There are also conflicting reports on cellular sources and subcellular location of IL-33 in the colonic mucosa, justifying a closer ... -
Mucosal cytokine profiles as biomarkers in inflammatory bowel disease -Translating molecular biology into clinical medicine
Rismo, Renathe (Doctoral thesis; Doktorgradsavhandling, 2013-01-11)Patogenesen ved inflammatorisk tarmsykdom (IBD) er fortsatt ikke fullstendig klarlagt. Mye forskning har blitt gjort de siste tjue årene for å kartlegge immunresponsen ved ulcerøs kolitt og Crohns sykdom. Man antar at et samspill mellom genetiske faktorer, miljøfaktorer og bakterier ligger bak utviklingen av sykdommen. Kartleggingen av immunresponsen i tarmen har vært grunnlaget for utviklingen av ...